TL;DR: A bunny hopping gait in cats often indicates joint or spinal discomfort and typically requires a scheduled vet visit; however, sudden onset or signs of severe pain require immediate emergency attention.
What does it mean if my cat has a bunny hopping gait?
A 'bunny hopping' gait in cats occurs when both hind legs move forward simultaneously rather than in a normal alternating sequence. While it might look cute or quirky, this movement pattern often indicates that the cat is attempting to minimize weight-bearing or joint rotation in the hips or knees. Common underlying causes include feline hip dysplasia, patellar luxation, or osteoarthritis. In some cases, it may also point to neurological issues or spinal discomfort.
Is it an emergency if my cat starts bunny hopping?
- The urgency level for a bunny hopping gait is generally Low if your cat is otherwise eating, drinking, and acting normally.
- This is typically a chronic condition that requires a scheduled veterinary appointment rather than an emergency visit.
- You should contact an emergency veterinarian immediately if the gait change occurred suddenly following a fall.
- Immediate care is required if your cat is crying out in pain, dragging their limbs, or is unable to stand.
How can capturing photos and videos help my veterinarian triage my cat?
- Capture videos at home where your cat is comfortable, as cats often hide discomfort or "freeze" in a clinical setting.
- Provide high-quality photos of your cat’s resting posture and clear videos of them running or climbing stairs.
- Visual evidence provides a realistic view of the musculoskeletal system in action, which is invaluable for a diagnosis.
- These recordings help the veterinarian differentiate between orthopedic pain and neurological weakness, leading to a faster and more accurate treatment plan.
Clinical Context (Merck Veterinary Manual)
In young cats, particularly puppies, a "bunny-hopping" gait characterized by paraparesis and weak or absent spinal reflexes in the pelvic limbs may indicate infection with Toxoplasma gondii or Neospora caninum. This often manifests between 3-8 weeks of age, potentially progressing to severe extensor rigidity, muscle atrophy, and ultimately, if untreated, dysphagia and fatal respiratory paralysis. Serum creatine kinase (CK) concentration is often elevated, and cerebrospinal fluid (CSF) analysis may reveal increased protein and leukocytes. Diagnosis can be aided by identifying serum or CSF antibodies or detecting the organism on muscle biopsy. Early treatment with clindamycin or trimethoprim/sulfadiazine and pyrimethamine may be beneficial.
Chapter: Neurology, Orthopedics
Source: The Merck Veterinary Manual, 11th Edition (Page 1096)